The myocarditis patients circulating spike samples are much earlier than the ones from the uninjured, but when you compare with ogata et al, it is much more in line.
The circulating spike hypothesis is falsifiable in multiple ways. The most obvious aren't antibodies actually doing the working. If Tcells are primed how can any amount of spike be produced given the speed of Tcell intervention at jab 2 and plus?
But more importantly w
How do spike bypass the vasculaf system's disseminative qualities?
It can't. Circulating spike issued should be in the tissue and in the vein, snd disseminated, ie systemic not in the aorta.
The Yonker report has a major problem.
The myocarditis patients circulating spike samples are much earlier than the ones from the uninjured, but when you compare with ogata et al, it is much more in line.
The circulating spike hypothesis is falsifiable in multiple ways. The most obvious aren't antibodies actually doing the working. If Tcells are primed how can any amount of spike be produced given the speed of Tcell intervention at jab 2 and plus?
But more importantly w
How do spike bypass the vasculaf system's disseminative qualities?
It can't. Circulating spike issued should be in the tissue and in the vein, snd disseminated, ie systemic not in the aorta.
Zn dit would not give rise to Tcell attacks.